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effects Overlapping here, however, are the tertiary zones for vision, hearing, and somatic sensation, the supramodal integration of which is essential to our awareness of space and person and certain aspects of language and calculation, as described below The parietal lobe is supplied by the middle cerebral artery, the inferior and superior divisions supplying the inferior and superior lobules, respectively, although the demarcation between the areas of supply of these two divisions is found to be variable Despite Critchley s pessimistic prediction that establishing a formula of normal parietal function will prove to be a vain and meaningless pursuit, our concepts of the activities of this part of the brain are now assuming some degree of order There is little reason to doubt that the anterior parietal cortex contains the mechanisms for tactile percepts Discriminative tactile functions, listed below, are organized in the more posterior, secondary sensory areas But the greater part of the parietal lobe functions as a center for integrating somatosensory with visual and auditory information in order to construct an awareness of one s own body (body schema) and its relation to extrapersonal space Connections with the frontal and occipital lobes provide the necessary proprioceptive and visual information for movement of the body and manipulation of objects and for certain constructional activities (constructional apraxia) Impairment of these functions implicates the parietal lobes, more clearly the nondominant one (on the right) Also, the conceptual patterns on which complex voluntary motor acts are executed depend on the integrity of the parietal lobes, particularly the dominant one Defects in this region give rise to ideomotor apraxia, as discussed further on The understanding of spoken and written words is partly a function of the supramarginal and angular gyri of the dominant parietal lobe as elaborated in Chap 23 The recognition and utilization of numbers, arithmetic principles, and calculation, which have important spatial attributes, are other functions integrated principally through these structures.

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function, is also predominantly the result of parietal lobe damage, and the relationship of the apraxias to language and to each other, exposes one of the most complicated issues in behavioral neurology The theoretical aspects of agnosia, particularly those related to the disturbances of visual processing, are discussed later in the chapter Cortical Sensory Syndromes The effects of a parietal lobe lesion on somatic sensation were rst described by Verger and then more completely by Dejerine, in his monograph L agnosie corti cale, and by Head and Holmes The latter, in their important paper of 1911, noted the close interrelationships between the thalamus and the sensory cortex As pointed out on page 133, the parietal postcentral cortical defect is essentially one of sensory discrimination, ie, an impairment or loss of the sense of position and passive movement and the ability to localize tactile, thermal, and noxious stimuli applied to the body surface; to distinguish objects by their size, shape, and texture (astereognosis); to recognize gures written on the skin; to distinguish between single and double contacts (two-point discrimination); and to detect the direction of movement of a tactile stimulus In contrast, the perception of pain, touch, pressure, vibratory stimuli, and thermal stimuli is relatively intact This type of sensory defect is sometimes referred to as cortical, although it can be produced just as well by lesions of the subcortical connections Clinicoanatomic studies indicate that parietocortical lesions that spare the postcentral gyrus produce only transient somatosensory changes or none at all (Corkin et al; Carmon and Benton) The question of bilateral sensory de cits as a result of lesions in only one postcentral convolution was raised by the studies of Semmes and of Corkin and their associates In tests of pressure sensitivity, two-point discrimination, point localization, position sense, and tactile object recognition, they found bilateral disturbances in nearly half of their patients with unilateral lesions, but the de cits were always more severe contralaterally and mainly in the hand These disturbances of discriminative sensation and the subject of tactile agnosia are discussed more fully in Chap 9 Dejerine and Mouzon described another parietal sensory syn drome in which touch, pressure, pain, thermal, vibratory, and position sense are all lost on one side of the body or in a limb This syndrome, more typically the result of a thalamic lesion, may also occur with large, acute lesions (infarcts, hemorrhages) in the central and subcortical white matter of the contralateral parietal lobe; in the latter case these symptoms recede in time, leaving more subtle defects in sensory discrimination Smaller lesions, particularly ones that result from a glancing blow to the skull or a small infarct or hemorrhage, may cause a defect in cutaneous-kinesthetic perception in a discrete part of a limb, eg, the ulnar or radial half of the hand and forearm; these cerebral lesions may mimic a peripheral nerve or root lesion (Dodge and Meirowsky) Also, a pseudothalamic pain syndrome on the side deprived of sensation by a parietal lesion has been described (Biemond) In a series of 12 such patients described by Michel and colleagues, burning or constrictive pain, identical to the thalamic pain syndrome (page 141), resulted from vascular lesions restricted to the cortex The discomfort involved the entire half of the body or matched the region of cortical hypesthesia; in a few cases the symptoms were paroxysmal Head and Holmes drew attention to a number of interesting points about patients with parietal sensory defects the easy fatigability of their sensory perceptions; the inconsistency of responses to painful and tactile stimuli; the dif culty in distinguishing more.

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