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Description This parameter is used to specify in list format the Web items whose navigational state you want to transfer to another Web template You use the Web Item (ITEM_REF) parameter to enter the various Web items Note that you must have already inserted all the Web items that you specify here in the current Web template This parameter is used to specify whether you want to use a new window to display the Web application in which you want to display a copy of the current navigational state of a data provider or Web item This command is used to set variable values at run time for a Web application Each of the variable values comes with its own list of values, including hierarchies, characteristic values, ranges, selection options, exclusion values, and inclusion values This parameter is used to specify whether the variable screen is to be displayed If you do not specify a value, the variable screen is displayed only if mandatory variables are not filled This parameter is used to enter a variant You can start a Web application by using a variant or you can fill the variable screen with the values from the variant This parameter is used to enter a list of individual variables You need to set the following parameters for each entry in the list: Variable (VARIABLE) Variable Type (VARIABLE_TYPE) You can choose from the following variable types: Input String for Variable (VARIABLE_INPUT_STRING) Characteristic Value Variable (INFO_OBJECT_MEMBER_VARIABLE) Hierarchy Variable (HIERARCHY_VARIABLE) Value Set Variable (VALUE_SET_VARIABLE) Text Variable (TEXT_VARIABLE) Formula Variable (FORMULAR_VARIABLE) Binding Type (SELECTION_BINDING_VARIABLE) Web Item with Manual Input (ITEM_INPUT) Depending on the variable type you select, you must set additional parameters This command is used to display the variable screen at run time for the Web application This command has no parameters This command is used to export the Web application in a format that is identified in the export process This can be an Excel document, PDF, or other formats This parameter is used to select the Web template that is to be used for the export.

in contrast to delirium, where the changes may be relatively minor Bilateral high-voltage slow waves in the range of two to four per second (delta) or ve to seven per second (theta) are the usual ndings These changes surely re ect one aspect of the central problem the diffuse impairment of the cerebral mechanisms governing alertness and attention In some ways, as noted earlier, the disorganized thinking and behavior of the confusional states may re ect the loss of integrated activity of the associative regions of the cortex

Clinical Features of Delirium These are most characteristically depicted in the patient undergoing withdrawal from alcohol after a sustained period of intoxication The symptoms usually develop

over a period of 2 or 3 days The rst indications of the approaching attack are dif culty in concentration, restless irritability, increasing tremulousness, insomnia, and poor appetite There may be momentary disorientation, an occasional inappropriate remark, or transient illusions or hallucinations One or several generalized convulsions may precede or initiate the delirium These initial symptoms rapidly give way to a clinical picture that, in severe cases, is one of the most colorful in medicine The patient is inattentive and unable to perceive all elements of his situation He may talk incessantly and incoherently and look distressed and perplexed; his expression may be in keeping with vague notions of being annoyed or threatened by someone From his manner and the content of his speech, it is evident that he misinterprets the meaning of ordinary objects and sounds, misidenti es the people around him, and is experiencing vivid visual, auditory, and

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Variable Screen (VARIABLE_SCREEN) Variant (VARIABLE_VARIANT)

tactile hallucinations, often of a most unpleasant type At rst the patient can be brought into touch with reality and may, in fact, identify the examiner and answer other questions correctly; but almost at once he relapses into a preoccupied, confused state, giving incorrect answers and being unable to think coherently As the process evolves the patient cannot shake off his hallucinations and is unable to make meaningful responses to the simplest questions and is, as a rule, profoundly disoriented The signs of overactivity of the autonomic nervous system, more than any others, distinguish delirium from all other confusional states Tremor of fast frequency and jerky restless movements are practically always present and may be violent Sleep is impossible or occurs only in brief naps Speech is reduced to an unintelligible muttering The face is ushed, the pupils are dilated, and the conjunctivae are injected; the pulse is rapid and soft, and the temperature may be raised There is excessive sweating After 2 or 3 days, the symptoms abate, either suddenly or gradually, although in exceptional cases they may persist for several weeks The most certain indication of the subsidence of the attack is the occurrence of lucid intervals of increasing length and sound sleep Recovery is usually complete In retrospect, the patient has only a few vague memories of his illness or none at all Delirium is subject to all degrees of variability, not only from patient to patient but in the same patient from day to day and even hour to hour The entire syndrome may be observed in one patient and only a few fragments in another In its mildest form, as often occurs in febrile diseases, the delirium consists of an occasional wandering of the mind and incoherence of verbal expression This form, lacking motor and autonomic overactivity, is sometimes referred to as a quiet or hypokinetic delirium and can hardly be distinguished from the confusional states described above Pathology and Pathophysiology of Delirium The brains of patients who have died in delirium tremens without associated disease or injury usually show no pathologic changes of signi cance Intoxication with a number of medications, particulalry those with atropinic effects, and certain abused drugs such as the hallucinogens cause a delirious state Delirium may also occur in association with a number of recognizable cerebral diseases, such as viral (herpes) encephalitis or meningoencephalitis, Wernicke disease, cerebral trauma, cerebral hemorrhage, or multiple embolic strokes due to subacute bacterial endocarditis, cholesterol or fat embolism, or following cardiac or other surgery The topography of the lesions in most of these conditions is of interest; they tend to be localized in the midbrain and hypothalamus and in the temporal lobes, where they involve the reticular activating and limbic systems Involvement of the hypothalamus perhaps accounts for the autonomic hyperactivity that characterizes delirium in these cases of gross cerebral disease That these are not the only sites implicated is emphasized by the observations that an acute agitated delirium has occurred at one time or another with lesions involving the fusiform and lingual gyri and the calcarine cortex (Horenstein et al); the hippocampal and lingual gyri (Medina et al); or the middle temporal gyrus (Mori and Yamadori) Electrical stimulation studies of the human cerebral cortex during surgical exploration and studies by positron emission tomography (PET) have emphasized the importance of the temporal lobe in the genesis of complex visual, auditory, and olfactory hallucinations Subthalamic and midbrain lesions may give rise to visual hallucinations that are not unpleasant and are accompanied by good insight ( peduncular hallucinosis of Lhermitte) For reasons.

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